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The lesions are associated with increased PDGFβ and TGFβ signaling that activate 1CNRS, UMR 7213, University of Strasbourg, 67401, Illkirch, France. 2Department of Molecular Genetics, University of Texas Southwestern Medical Center, Dallas, TX, USA.

*Present address: Simmons Comprehensive Cancer Center, 5325 Harry Hines Blvd Y4.206, UT Southwestern Medical Center, Dallas, Texas. †On leave from the Institute of Chemistry, Chinese Acad- emy of Sciences, Beijing, China.

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The University of Texas Southwestern Medical Center

The University of Texas Southwestern Medical Center

The University of Texas Southwestern Medical Center

E-mail: walter.hu@utdallas.edu; Tel: +1 972 883 6329 bDepartment of Pharmacology, Simmons Comprehensive Cancer Center, University of Texas, Southwestern Medical Center, Dallas, TX, 75390, USA cDepartment of Mechanical Engineering and Mechanics Bioengineering Program, Lehigh University, USA † Electronic

Department: Center for Alzheimer’s and Neurodegenerative Diseases, Neurology Challenge Tau deposition in fibrillar aggregates is linked to myriad neurodegenerative diseases, and underlies dementia in Alzheimer’s Disease (AD) and Chronic Traumatic Encephalopathy (CTE).

The University of Texas Southwestern Medical Center